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Blood flow was measured prior to this exposure and following a cooling period. Researchers discovered that TRPA1 acted in two distinct ways – first by sensing the switch in temperature, and then by stimulating the defensive constriction of blood vessels. The subsequent vasodilatation stage was also reliant on TRPA1 activation and was essential for restoring blood circulation. Related StoriesScalable production of gene therapy vectors: an interview with Frank UbagsStudy suggests potential new way to block cancer-leading to geneAngina in women linked with abnormal heart bloodstream flowAlthough an early on study, the results offer a new understanding of TRPA1's role in cold publicity and provide impetus for further study into how this gene could be targeted to improve the body's protective response to cold.You will see the entire Kaiser Daily Health Policy Survey, search the archives, or join email delivery of in-depth coverage of wellness policy developments, discussions and debates. The Daily Health Policy Report is released for Kaisernetwork.org, a free of charge support of The Henry J. Kaiser Family Foundation. Copyright 2009 Advisory Board Firm and Kaiser Family Basis. All rights reserved.. The Kiva Program can be a novel implant-based answer for vertebral augmentation and a departure from balloon kyphoplasty , rendering it the first fresh approach to the treating vertebral compression fractures in over ten years.

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